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Researchers Database

You have entered the IISP: Interactive Information System on Pathogenomics

 

If you would like to insert information about your research or research group, please click to the following link to download the form. After filling it out, please send back to m.karrasch@fz-juelich.de or g.gebreselassie@fz-juelich.de.

Here you will find information about Research groups in the PathoGenoMics fields from the ERA-NET partner countries (Austria, Finland, France, Hungary, Israel, Latvia, Portugal, Slovenia and Spain).
This information is supposed to support cooperation between researchers from different European countries
and thus enhance the development of a European Research Area for PathoGenoMics .

 

The following information is available and can be searched for:

  • researcher names
  • Institution of the respective researcher, city and country of his/her institution
  • Contact data of the researcher (address, phone, email)
  • Research topics and studied microorganisms of the researcher
  • Special techniques applied by the researcher
  • Potential cooperation topics suggested by the researcher

If you have any comments/questions or if you would like to add some information, please contact m.karrasch@fz-juelich.de

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Name: Dr. Viljanen, Matti
Address: Kiinamyllynkatu 10
Institution: "Department of Medical Microbiology University of Turku"
 
City: Turku Zip: FI-20520
Country: Finland Phone:
    Fax:
Email: matti.viljanen@utu.fi
www:

 

Research Topics:
Borreliae, borreliosis, Lyme disease, borrelia arthritis, immune evasion, neutrophil, dendritic cell, mouse model, CD38, TNF-alfa Our first aim is to understand the mechanisms by which borrelia evades the first line immune defence and disseminates causing a multi-organ infection. Our second aim is to clarify the mechanisms of persistent and treatment resistant manifestations of LB that may lead to longstanding disability of the patients. First hypothesis is that borrelia interferes with dendritic cells (DCs) that are important operators in the initiation and regulation of immune response. Second hypothesis is that borrelia infection becomes latent during antibiotic treatment and borrelia resides in a dormant form in host tissues.

 

Organisms studied:
  • Borrelia
  • Borrelia burgdorferi

 

Special methods / technologies:
Gene arrays, flow cytometry, fluorescence imaging, PET-scanning, production of monocyte-derive dendritic cells, production of borrelia mutants, mouse model for chronic Lyme borreliosis

 

Suggestions for potential research cooperations:
Whole genome array of Borrelia burgdorferi
Ultrasensitive detection techniques for microbes or theis components in tissues

 
   
   
   
     
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